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Study Pinpoints Immune Signals Behind Rare Myocarditis After mRNA COVID Vaccines

Preclinical work ties CXCL10 plus interferon gamma to cardiac injury, pointing to cytokine blockers or genistein as possible mitigations.

Overview

  • A Stanford-led team reported elevated CXCL10 and interferon gamma in vaccine recipients with myocarditis and reproduced this cytokine surge in human immune cells and inoculated mice.
  • Human cardiac spheroids exposed to these signals showed impaired contraction, and vaccinated mice had reduced cardiac injury markers when the cytokines were blocked with antibodies.
  • The estrogenlike compound genistein prevented or reversed damage in mice and cell models, and the authors said it did not diminish vaccine effectiveness in their experiments.
  • Surveillance data indicate the condition remains uncommon, with about 7 cases per million after first doses, higher rates after second doses, and the greatest incidence in men under 30.
  • Experts emphasize that SARS‑CoV‑2 infection causes myocarditis more frequently and often more severely, and they note the new findings are preclinical with mouse studies using relatively high doses.