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Bacterial RNA-Repair System Identified as New Target to Boost Common Antibiotics

The Nature Communications study shows cell-to-cell differences in an Rtc pathway help some E. coli withstand treatment.

Overview

  • University of Edinburgh researchers uncovered how a bacterial Rtc system repairs damaged RNA to sustain protein production under antibiotic stress.
  • The work combines computational modelling with laboratory experiments in E. coli to map how this repair pathway preserves growth during drug exposure.
  • Expression of the Rtc system varies between individual cells, creating unpredictable treatment responses that can leave subsets of bacteria alive.
  • The authors propose developing inhibitors of key Rtc components to enhance the potency of existing antibiotics, noting the concept remains preclinical.
  • Findings were published in Nature Communications with contributions from Queen Mary University of London and Imperial College London, supported by BBSRC, the Leverhulme Trust and Wellcome.